Insufficient Evidence for "Autism-Specific" Genes



Polysorbate 80: A Risky Vaccine Ingredient

Myalgia and chronic fatigue syndrome following immunization

Engineering squalene analogues for novel vaccine adjuvant emulsions

Aluminum hydroxide injections lead to motor deficits and motor neuron degeneration

Administration of aluminium: adverse long term neurological outcomes

Our previous ecological studies of autism spectrum disorder (ASD) has demonstrated a correlation between increasing ASD rates and aluminium (Al) adjuvants in common use in pediatric vaccines in several Western countries.

We provide an animal model to explore potential behavioural phenotypes and central nervous system (CNS) alterations using s.c. injections of Al hydroxide in early postnatal CD-1 mice of both sexes.

Injections of a "high" and "low" Al adjuvant levels were designed to correlate to either the Scandinavian or US pediatric vaccine schedules vs. control saline-injected mice.

Both male and female mice in the "high Al" group showed significant weight gains following treatment up to sacrifice at 6 months of age.

These current data implicate Al injected in early postnatal life in some CNS alterations that may be relevant for a better understanding of the aetiology of ASD.

Long-term toxicity of a Roundup herbicide

A vaccine adjuvant is an agent 'meant' to stimulate the immune system and 'increase' the response to a vaccine.

Aluminium adjuvants are used most often but adjuvants like squalene and new organophosphates are being tested.

1928   Staphylococcus infection kills 12 of 21 children inoculated with a diphtheria vaccine that lacked a preservative.

Preservatives available bur not standardly used in this period of time include thimerosal, phenoxyethanol, and formaldehyde.

Phenoxyethanol, a glycol ether, is toxic to the kidneys, nervous system, and liver, and repeated, long-term exposure can cause organ damage.

Phenoxyethanol is synthesized by treating phenol with ethylene oxide in an alkaline medium.

Phenoxyethanol along with parabens; sulfates; phthalates; polyethylene; propylene glycol and butylene glycol in comestics should be avoided.

The Japan and EU consider phenoxyethanol safe for use as a preservative with a maximum concentration of 1.0%.

FDA issued a public warning revealing that the ingestion of phenoxyethanol can be toxic and harmful for infants when it was found in a nipple cream called Mommy's Bliss.

Aluminium in brain tissue in autism

Neurodevelopmental disorders: thimerosal immunizations

'ASIA' - autoimmune/inflammatory syndrome induced by adjuvants


"Human oil adjuvants are the most insidious chemical weapon ever devised. Squalene is a trigger for a real biological weapon - what Soviet researchers called a biological time bomb." - Gary Matsumoto

Squalene, an oil molecule native to the human body, is found throughout the nervous system and the brain.

Squalene is also the biochemical precursor to the whole family of steroids.

Squalene adjuvants are key ingredients in the next generation of vaccines intended for mass immunization around the globe.

Squalene galvanizes the immune system into attacking the nervous system.

1970s Michael Whitehouse and Frances Beck inject squalene oil combined with other materials into rats and find that few oils are more effective at causing the animal versions of arthritis and multiple sclerosis.

There is a close match between the squalene-induced diseases in animals and those observed in humans injected with this oil:

rheumatoid arthritis, multiple sclerosis and systemic lupus erythematosus.

Cannabinoids inhibit neurodegeneration in models of multiple sclerosis

Clinical Use of Cannabinoids for Symptom Control in Multiple Sclerosis

Changes Uncovered in Gut Bacteria of People with Multiple Sclerosis

Digesting squalene isn't a problem.

Injecting squalene into the blood galvanizes the immune system to attack.

Autoimmune disorders occur because the body identifies the squalene molecules as having breached system barriers - they are out-of-place.

Once the body identifies squalene molecules as an enemy a self-destructive cross reaction may take place against the same molecule where it occurs naturally in the body - where it is critical to the health of the nervous system.

Once self-destruction begins, it doesn't stop as the body keeps making the molecule that the immune system is trained to attack and destroy.

"There are now data in more than two dozen peer-reviewed scientific papers, from ten different laboratories in the US, Europe, Asia and Australia, documenting that squalene-based adjuvants can induce autoimmune disorders in animals - observed in mice, rats, guinea pigs and rabbits.

Sweden's Karolinska Institute has demonstrated that squalene alone can induce the animal version of rheumatoid arthritis.

The Polish Academy of Sciences has shown that in animals, squalene alone can produce catastrophic injury to the nervous system and the brain.

The University of Florida Medical School has shown that in animals, squalene alone can induce production of antibodies specifically associated with systemic lupus erythematosus." - Dr. Johnny Lorentzen, Karolinska Institute Sweden

"Squalene contributed to the cascade of reactions called "Gulf War syndrome" - arthritis, fibromyalgia, lymphadenopathy, rashes, photosensitive rashes, malar rashes, chronic fatigue, chronic headaches, abnormal body hair loss, non-healing skin lesions, aphthous ulcers, dizziness, weakness, memory loss, seizures, mood changes, neuropsychiatric problems, thyroid malfunction, anemia, elevated ESR (erythrocyte sedimentation rate), systemic lupus erythematosus, multiple sclerosis, Lou Gehrig's disease, Raynaud's phenomenon, Sjörgren's syndrome, chronic diarrhea, night sweats and low-grade fever." - micropaleontologist Dr. Viera Scheibner

Maneb | C4H6MnN2S4

Zineb | C4H6N2S4Zn

Maneb and zineb fungicides in microagroecosystem

"The tomatoes were peeled by hand and only the peels were analyzed."

2000 David Ronald Brown advanced research related to the role of metals in the cause of prion diseases such as vCJD.

Media attention focused on this work when it became associated with that of the farmer Mark Purdey, who argued that human cases of vCJD might be caused by exposure to the systemic organophosphate insecticide Phosmet, used as a dip to contain warble fly, and manganese in the salt lick.

"Many might be surprised to hear that organophosphates were developed by Nazi chemists during the course World War Two, as a chemical weapon nerve agent.

One formulation of the insecticide - Maneb, or Mancozeb - actually contains manganese in addition to organophosphate.

Phosmet organophosphate has been used at high doses in British warble fly campaigns.

In 1996, ICI subsidiary Zeneca sold the phosmet patent to a PO Box company in Arizona called Gowan -just one week before the UK government admitted to a link between BSE and nvCJD.

The politically well-connected British pharmaceuticals group, ICI has the financial and political clout to block research into any cause other than the infective model.

Indeed no substantive alternative research has been done.

British BSE disease management and research bodies have taken decisions that do not seem guided by spirited scientific enquiry.

Mysterious prions that jump species is a preferred research arena.

Scientist and organic farmer, Mark Purdey gave evidence to the UK BSE inquiry, that warble fly insecticide was the cause of the disease.

The scientist wheeled out to rubbish Purdy's evidence - Dr. David Ray, later turned out to have been receiving funding from the insecticide manufacturer ICI.

A lobby group that includes Bayer, Monsanto, Novartis, Pfizer, Roche and Schering-Plough was behind the effort to discredit Purdey.

In December 1999, the same David Ray was appointed to the UK Veterinary Products Committee (VPC) -a government body that licences animal medicines." - Fintan Dunne

Organophosphate can deform the prion molecule, rendering it ineffective at buffering free radicals.

These prion are then partial bond with manganese and become 'rogue' prion.

A chain reaction where by rogue prions turn others to rogues explains the bovine spongiform disease mechanism.

Brown showed how prion protein bonds benignly with copper, but lethally with manganese.

Even natural variations in relative environmental availability of manganese versus copper can trigger prion degradation.

Consequences of manganese replacement of copper for prion protein function and proteinase resistance

Cannabis Kills MRSA, Disrupts Prion Diseases

COVID-19 RNA Based Vaccines and the Risk of Prion Disease

2009 "Some adjuvants may exert adverse effects upon injection or, on the other hand, may not trigger a full immunological reaction.

The mechanisms underlying adjuvant adverse effects are under renewed scrutiny because of the enormous implications for vaccine development.

The ability of the immune system to recognize molecules that are broadly shared by pathogens is, in part, due to the presence of special immune receptors called toll-like receptors (TLRs) that are expressed on leukocyte membranes.

The very fact that TLR activation leads to adaptive immune responses to foreign entities explains why so many adjuvants used today in vaccinations are developed to mimic TLR ligands." - Adjuvants and autoimmunity

2015 "Since vaccines are given to previously healthy hosts, who may have never developed the disease had they not been immunized, adverse events should be carefully accessed and evaluated even if they represent a limited number of occurrences." - Vaccines, adjuvants and autoimmunity

2016 "A previously healthy 36-year-old female presented with clinical symptoms of thyrotoxicosis including tachycardia, anxiety, and tenderness in her neck .

One month before her presentation, she received H1N1 vaccine.

Thyroid function tests confirmed remarkable thyrotoxicosis." - Autoimmune Disorder Induced by Adjuvants and Thyroid Autoimmunity

2017 "Autoimmune disorder induced by adjuvants (ASIA) has been widely described in many studies conducted thus far." - Autoimmune disorder induced by adjuvants

March 27, 2018 "Incidence of autoimmune disorder has been rising at an enormous rate.

A study indicates that a significant factor in causing them may be the common bakers or brewers yeast, Sacccharomyces cerevisiae used in many vaccinations, including HepB, which is given to nearly all newborn babies in the United States before they're a day old.

The specific part of S. cerevisiae that's of concern is mannan, which is found in the cell walls of yeasts and also in mammalian glycoproteins.

Glycoprotein is found in cell walls, connective tissues like collagen, gastrointestinal mucous secretions, and blood plasma.

Glycoproteins perform many functions.

Obviously, if the immune system goes on the attack against mannan or polysaccharide, it can be devastating.

Yet, that appears to be happening in many autoimmune diseases." - Heidi Stevenson, Yeast in Vaccines Tied to Autoimmune Diseases

Glycoproteins contain oligosaccharide chains covalently attached to amino acid side-chains.

The carbohydrate is attached to the protein in a cotranslational or posttranslational modification.

This process is known as glycosylation.

Secreted extracellular proteins are often glycosylated.

Anti-Saccharomyces cerevisiae antibodies in inflammatory bowel disease

Anti-Saccharomyces as a Biomarker in Children With Crohn Disease

Prevalence anti-saccharomyces cerevisiae in primary Sjögren's syndrome

Anti-Saccharomyces cerevisiae with Anti-ß2 Glycoprotein I antibodies

Anti-Saccharomyces cerevisiae as antibodies in autoimmune hepatitis

"There is an urgent need to start adequately adjusted epidemiological studies to obtain better evidence which percentage of patients develop symptoms of autoimmune disorders after implant surgery." - ASIA; Shoenfeld's syndrome: A new flame.

unique library index

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